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While the classical EDS is part of the disease group A in which collagen structure and collagen processing is affected resulting in the main clinical features of skin hyperextensibility and joint hypermobility, EDS entities of other genetic / pathogenic groups have a wider phenotypic variability

We recheck symptoms and labs (especially homocysteine) and adjust

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Glutathione mutants showed also a higher susceptibility to SFN treatment To address the question if the more susceptible phenotype of glutathione mutants towards AITC was unique for this GHP, sulforaphane (SFN), an aliphatic ITC possessing a longer side chain than AITC, was tested

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